Honestly I cannot bear non-euphoric stims, it's like, where's the happiness man? it doesn't motivate me if I don't have any euphoria, it's so cold it's boring as fuck
Cold dopamine is just a way to procrastinate more, having ADHD, so I consider it to be rubbish even for adhd...
...
for me less boring = Euphoria, there's no other way.
Task-starting and sustained attention are different beasts for sure. When I was finishing my dissertation, dextroamphetamine definitely provided both, along with some of the mood lift, bordering on euphoria. The mood lift makes starting tasks less daunting, and the tunnel vision/hyperfocus help keep effort consistent.
Are you saying you think 2-FMA is worse for you because it's a stronger dopamine releaser? My understanding has been, lacking actual binding profile studies, racemic 2-FMA is probably actually a stronger NE releaser than
d-amph, which in turn is stronger than
d-meth. I haven't seen anyone even speculate as to enantiomer-specific activity of 2-FMA, but pulling things out of thin air, I would guess, as with meth, the
l-enantiomer of 2-FMA is extremely biased towards NE, and the
d-enantiomer is more balanced. This would explain the apparent ceiling effect of 2-FMA. Since reports are that high doses increase vasoconstriction and tachycardia without more central stimulation, it would make sense that racemic 2-FMA, which as far as I know is all anyone has ever taken (correct me if I'm wrong), would be NE biased as a single drug. 2-FMA is also less associated with psychosis and mania in user reports, which suggests less dopamine bias. This could all be complicated by metabolism—there's a paper on plausible metabolites of 2-FMA, but as with its binding potency, the literature is sparse.
I do agree that dopamine release is not strictly better for ADHD pharmacotherapeutics. Most anecdotes I've read about attempts to use
d-meth for ADHD suggest that it can be counterproductive, but more so
because of the heightened euphoria. I view mood lift as something that needs to be optimized: enough to overcome task anxiety and increase task enjoyment, but not so much you stop worrying at all about the actual life obligation ADHD is interfering with. I also personally wonder if the apparent anxiolysis along with more subtle stimulation of 2-FMA might be due to interactions with sigma receptors, or maybe a relative lack thereof compared to meth. Since 2-FMA is unlikely to be enough of a cash cow to merit clinical trials, and hasn't been the subject of alarmist media reporting, I think the motivation to do binding assays on it is weak, and so insight about its pharmacology is likely to continue only at a trickle.
Good luck in any case with your research and writing. It's not the easiest thing for those of us with ADHD diagnoses, and optimizing medication goes a long way to help with that.