woodyfence
Bluelighter
- Joined
- May 31, 2016
- Messages
- 81
I have ADHD and have found natural inhibiting mechanisms inherited from my mother's singular alcohol and tobacco use during pregnancy. I have found myself to inhibit NMDA, PCP2, and several cholinergic mechanisms in my body throughout my 20 years of being alive and using amphetamine to medicate. I've found several papers which indicate these mechanisms could be used to inhibit STD viruses and cancer cell growth. Here is research evidence ithat inhibitory responses in my biology could be used for STD and cancer research:
Cancer:
www.ncbi.nlm.nih.gov
pubmed.ncbi.nlm.nih.gov
Glutamate inhibition prevents cancer spread
STD:
EZH1 and EZH2 mechanisms are related to the histamine system (HX) which I likely naturally inhibit
www.niaid.nih.gov
I've been infected with an unknown STD, quit my job, and all attempts to remedy this situation through finding a sustainable job have been shut down as my surroundings are filled with liars. Here's my entire life story autobiography with about 140,000 words going into even more detail of the situation wherein I only have enough money for about 2-3 weeks of food. It also details the results of my recent "hunger strike" where I started tripping NMDA balls with red slashes appearing everywhere as my cells were sacrificed and later rebuilt. If you'd like to contribute to me, that would be greatly appreciated as I intend to reciprocate to the community at large. I'm a software engineer seeking a sustainable life outside the shortsightedness in my current life.
github.com/josephcantrell14/journal
Cancer:
Inhibitors of glutamate release from breast cancer cells; new targets for cancer-induced bone-pain
Glutamate is an important signaling molecule in a wide variety of tissues. Aberrant glutamatergic signaling disrupts normal tissue homeostasis and induces several disruptive pathological conditions including pain. Breast cancer cells secrete high levels ...
www.ncbi.nlm.nih.gov
Inhibition of glutamate oxaloacetate transaminase 1 in cancer cell lines results in altered metabolism with increased dependency of glucose - PubMed
Our study suggests an important role of GOT1 to coordinate the glycolytic and the oxidative phosphorylation pathways in KRAS mutated cancer cells. GOT1 is crucial to provide oxaloacetate at low glucose levels, likely to maintain the redox homeostasis. Our data suggest GOT1 as a possible target...
pubmed.ncbi.nlm.nih.gov
STD:
The page you’re looking for isn’t available
It's possible that the page is temporarily unavailable, has been moved, renamed, or no longer exists. Here are some suggestions to find what you are looking for:
I've been infected with an unknown STD, quit my job, and all attempts to remedy this situation through finding a sustainable job have been shut down as my surroundings are filled with liars. Here's my entire life story autobiography with about 140,000 words going into even more detail of the situation wherein I only have enough money for about 2-3 weeks of food. It also details the results of my recent "hunger strike" where I started tripping NMDA balls with red slashes appearing everywhere as my cells were sacrificed and later rebuilt. If you'd like to contribute to me, that would be greatly appreciated as I intend to reciprocate to the community at large. I'm a software engineer seeking a sustainable life outside the shortsightedness in my current life.
github.com/josephcantrell14/journal